Sökning: "Elisabeth Karlsson"
Visar resultat 6 - 10 av 12 avhandlingar innehållade orden Elisabeth Karlsson.
6. Praktiken im Zwischenraum : Transitorisches Schreiben bei Katja Lange-Müller
Sammanfattning : This thesis analyzes works by the contemporary German author Katja Lange-Müller, focusing on their representations of borders and in-between spaces. Thus, the thesis offers a new perspective on Lange-Müller’s work, which, in the few previous studies, has mainly been interpreted within the context of ‘subversive writing’ in the German Democratic Republic in the 1980s. LÄS MER
7. Från slutna institutioner till institutionaliserat omhändertagande
Sammanfattning : In recent years, a number of reforms have been made regarding the provision of care to people experiencing psychiatric disabilities. The aim of these reforms was to enable these people to participate in society and to ”live as others”. LÄS MER
8. Swedish maternal health care in a multiethnic society - including the fathers
Sammanfattning : Preventive work in maternal and child health care has a long history in Sweden. Today, Sweden has achieved the lowest maternal and child mortality rates globally based on a maternal health care system regulated by national recommendations; offered to every woman, free of chare, on a continuity basis, by registered midwives at municipal clinics within the community with the purpose of being assessable for all women. LÄS MER
9. Metabolic and Endocrine Response in the Acute Stage of Subarachnoid Hemorrhage
Sammanfattning : The rupture of an aneurysm in subarachnoid hemorrhage (SAH) is a dramatic event causing a severe impact on the brain and a transient or permanent ischemic condition. Several types of responses to meet the challenges of SAH have been found in the acute phase, including activation of the hypothalamic-pituitary-adrenal (HPA) axis and the sympathetic nervous system, elevated levels of brain natriuretic peptide (BNP), and disturbances in cerebral and systemic metabolism. LÄS MER
10. Diabetes in Young Adults : Remission, β-cell function and markers of inflammation
Sammanfattning : Type 1 diabetes is caused by immuno-mediated β-cell destruction leading to insulin deficiency and hyperglycaemia. The decline in β-cell function and the clinical course after diagnosis vary. Whether the process of destruction of the β-cells is associated with markers of a non-specific inflammatory response is unknown. LÄS MER