Sökning: "YopH"
Visar resultat 6 - 10 av 10 avhandlingar innehållade ordet YopH.
6. Yersinia-phagocyte interactions during early infection
Sammanfattning : Pathogenic Gram-negative Yersinia species preferentially target and inactivate phagocytic cells of the innate immune defense by translocation of effector Yersinia outer proteins (Yops) into the cells via a type III secretion system. This indicates that inactivation and avoidance of the early innate immune response is an efficient way for Yersinia species to avoid elimination and to cause diseases ranging from mild gastroenteritis (Y. LÄS MER
7. Temperature-inducible and calcium-regulated proteins encoded by the virulence plasmid of Yersinia
Sammanfattning : The pathogenic members of the genus Yersinia, Y. pseudotuberculosis, Y. pestis and Y. enterocolitica are transmitted from animals to man and may give rise to disease with a variety of symptoms. LÄS MER
8. Interaction between gastric pathogen Helicobacter pylori and host cells
Sammanfattning : H. pylori plays a causative role in the pathogenesis of gastritis, gastric atrophy, gastric and duodenal ulcer, and is believed to predispose to gastric adenocarcinoma as well as low-grade B-cell lymphoma. Lewisb (Leb) blood group antigen is a receptor for Leb adhesin-positive H. pylori strains. LÄS MER
9. Host-pathogen interactions during Campylobacter and Yersinia infections
Sammanfattning : The innate immune system is known for protecting the host against invading pathogens, for instance enteropathogens infecting the gastrointestinal tract. The production of e.g. antimicrobial peptides, cytokines, and chemokines by innate immune cells and intestinal epithelial cells contribute to bacterial clearance. LÄS MER
10. Multiple functions of YopN in the Yersinia pseudotuberculosis type III secretion system : from regulation to in vivo infection
Sammanfattning : The type 3 secretion systems (T3SSs) are virulence mechanisms used by various Gram-negative bacteria to overcome the host immunity. They are often target-cell contact induced and activated. Activation results in targeting of virulence effector substrates into host cells. LÄS MER