Sökning: "Herpes virus type 2"
Visar resultat 11 - 15 av 38 avhandlingar innehållade orden Herpes virus type 2.
11. ETHICS AND PUBLIC HEALTH INTERVENTIONS
Sammanfattning : Programmatic vaccination and screening are the reasons for this thesis. The practice of public health incorporating medicine’s cognitive contents along with contributions from other fields of human activities is basically a moral enterprise. LÄS MER
12. Protein recognition domains in heparan sulfate
Sammanfattning : Heparan sulfate (HS) is a sulfated glycosaminoglycan (GAG) implicated in various physiological and pathological processes such as cell proliferation, viral infection and inhibition of blood coagulation. These effects are due to interactions of HS with proteins. LÄS MER
13. Diagnosis in aseptic meningitis and immune response in herpes simplex virus infections
Sammanfattning : Herpes simplex virus type 1 and 2 are ubiquitous and appear often asymptomatic but some individuals suffer from recurrent infections and the causes are largely unknown. In this thesis I present results from evaluations of diagnostic and etiologic studies of methods used for the detection of acute aseptic meningitis (AAM) and in HSV-2 meningitis in particular. LÄS MER
14. Immunization approaches and molecular signatures for mucosal immunity to primary and recurrent genital herpes
Sammanfattning : Genital herpes is most commonly caused by herpes simplex virus type 2 (HSV-2), and is a prevalent sexually transmitted infection worldwide. Despite numerous efforts, there is currently no licensed vaccine against the disease. LÄS MER
15. Studies on host-related pathogenesis of herpes simplex type-1 encephalitis in rat
Sammanfattning : In order to explore the molecular mechanisms of Herpes simplex encephalitis (HSE), a severe infection of the central nervous system (CNS) caused by Herpes simplex type 1 virus (HSV-1); a rat model resembling the human condition was characterized in the DA (Dark Agouti) strain. After injection into the whiskers’ area HSV-1 entered the CNS at the level of the brain stem via the trigeminal ganglion, subsequently spreading to the thalamus, cortex and olfactory bulb, leading to death at five days post infection (dpi). LÄS MER