Sökning: "symptom provocation"

Visar resultat 16 - 20 av 22 avhandlingar innehållade orden symptom provocation.

  1. 16. Immunological and inflammatory mechanisms in ocular allergy with special reference to vernal keratoconjunctivitis : clinical and experimental studies

    Författare :Per Montan; Karolinska Institutet; Karolinska Institutet; []
    Nyckelord :allergic conjunctivitis; allergen challenge; atopy; conjunctival biopsy; eosinophils; ECP; IgE; immunohistochemistry; inflammation; interleukins; T-cells; tears; vernal keratoconjunctivitis;

    Sammanfattning : Ocular allergy encompasses a wide range of conditions, most of which implicate IgE in the pathogenesis. In contrast to the benign nature of seasonal allergic conjunctivitis (SAC), the less common chronic eye diseases vernal and atopic keratoconjunctivitis (VKC and AKC) are severe and debilitating disorders. LÄS MER

  2. 17. Toll-like Receptors in Allergic Rhinitis

    Författare :Mattias Fransson; Malmö Institutionen för kliniska vetenskaper; []
    Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; neutrophils; poly I:C ; uteroglobin; Toll-like receptors; Medicine human and vertebrates ; Medicin människa och djur ; Otorhinolaryngology; audiology; auditive system and speech; Otorinolaryngologi; audiologi; hörsel- och talorganen; innate immunity; LPS; allergic rhinitis; bone marrow;

    Sammanfattning : Allergic rhinitis is an inflammatory disorder with the characteristic symptoms itching, sneezing, secretion and blockage upon allergen contact. In addition to the local inflammation in the nose, there is a systemic component that influences peripheral blood, bone marrow and lungs. LÄS MER

  3. 18. Asthma, chronic bronchitis, chronic obstructive pulmonary disease (COPD) and respiratory symptoms among adults in Estonia : prevalence and risk factors : comparison with Sweden and Finland : the 'FinEsS' studies Estonia I

    Författare :Mari Meren; Karolinska Institutet; Karolinska Institutet; []
    Nyckelord :Epidemiology; asthma; chronic bronchitis; COPD; Estonia; the FinEsS Studies;

    Sammanfattning : The thesis is based on original research data of the Estonian part of the "FinEsS" studies, which are cross-sectional comparative studies between Finland, Estonia and Sweden aiming to estimate the prevalence of respiratory symptoms, asthma, chronic bronchitis, COPD and allergy and to assess risk factors for the diseases and conditions. In Estonia the study areas included Tallinn, the capital of the country, Saaremaa, an agricultural island in the Baltic Sea, and Narva, a heavily industrialised town located at the Russian border. LÄS MER

  4. 19. Quality of life and markers of inflammation : a study of asthma in primary care

    Författare :Per Olof Ehrs; Karolinska Institutet; Karolinska Institutet; []
    Nyckelord :Mild Asthma; primary care; quality of life; airway inflammation; bronchial responsiveness; fluticasone.;

    Sammanfattning : Asthma is a chronic disease that usually is diagnosed and treated in primary care. Most patients have a mild to moderate disease. Asthma is considered to be caused by inflammation of the airways. LÄS MER

  5. 20. Tryptophan-related Neurotransmission in the Brain: Disturbances Associated with Experimental Hepatic Encephalopathy

    Författare :Peter B F Bergqvist; Avdelningen för klinisk kemi och farmakologi; []
    Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; tetrodotoxin; serotonin; release; rat; quinolinic acid; potassium; chloroamphetamine; noradrenaline; neurotransmission; microdialysis; hepatic encephalopathy; fenfluramine; drug safety; dopamine; coma; citalopram; ammonia; calcium; brain; tryptophan; Pharmacological sciences; pharmacognosy; pharmacy; toxicology; Farmakologi; farmakognosi; farmaci; toxikologi;

    Sammanfattning : In the present study, L-tryptophan (TRP)-related disturbances in the brain in experimental hepatic encephalopathy (HE) were investigated. The endogenous NMDA-receptor agonist and L-TRP metabolite quinolinic acid (QUIN) has been suggested to be involved in the pathogenesis of HE. LÄS MER