Sökning: "N. Johan Persson"

Hittade 3 avhandlingar innehållade orden N. Johan Persson.

  1. 1. Models of the Distribution of Persistent Organic Pollutants in the Marine Environment

    Författare :N. Johan Persson; Dag Broman; Kevin Jones; Stockholms universitet; []
    Nyckelord :NATURVETENSKAP; NATURAL SCIENCES; persistent organic pollutants; mass balance models; fugacity models; eutrophication; PCDD F; PCB; PCN; soot carbon.; Environmental chemistry; Miljökemi;

    Sammanfattning : Persistent organic pollutants (POPs) is a group of chemicals that are toxic, undergo long-range transport and accumulate in biota. Due to their persistency the distribution and recirculation in the environment often continues for a long period of time. LÄS MER

  3. 2. On the role of morphometry in coastal ecosystem modelling and management

    Författare :Johan Persson; Uppsala universitet; []
    Nyckelord :NATURVETENSKAP; NATURAL SCIENCES; Earth sciences; Geovetenskap; Earth sciences; Geovetenskap; Sedimentology; sedimentologi;

    Sammanfattning : Many signs show that the Baltic Sea is increasingly eutrophicated. Primary production hasincreased significantly resulting in gradually decreasing deep water oxygen concentrationsduring the twentieth century. In the coastal zone, problems are often manifested as oxygendepletion resulting in extinction of macrobenthic fauna. LÄS MER

  4. 3. Inflammasome polymorphisms and the Inflammatory Response to Bacterial Infections

    Författare :Berhane Asfaw Idosa; Eva Särndahl; Anne Kelly; Bo Söderquist; Alexander Persson; Johan Bylund; Örebro universitet; []
    Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; Bacteremia; Cytokines; Gene variants; Inflammasome; Inflammation; Innate immunity; Neutrophils; Meningitis; Neisseria meningitidis; Biomedicin; Biomedicine;

    Sammanfattning : NLRP3 inflammasome; a key component of the innate immune system, can be activated by a number of pathogens and other threats of the body. Activation of the NLRP3 inflammasome triggers caspase-1 mediated maturationof IL-1β and IL-18. LÄS MER