Avancerad sökning
Visar resultat 1 - 5 av 10 avhandlingar som matchar ovanstående sökkriterier.
1. Malassezia-related diseases
Sammanfattning : The purpose of this thesis is to investigate whether the new taxonomy of Malassezia may have important clinical implications. In particular, the studies look at pityriasis versicolor and other dermatological disorders associated with Malassezia yeasts to see whether there is a clear association between the yeast species and body site colonized and/or disease state. LÄS MER
2. Atopic dermatitis in adults: prognosis and factors of importance for persistence
Sammanfattning : Atopic dermatitis (AD) is a common skin disease with multifactorial aetiology. Elevatedlevels of serum IgE are common in AD patients and IgE antibodies to the yeast Malassezia, amember of the cutaneous microflora, are also common. LÄS MER
3. Studies on colonization and infection with Staphylococcus aureus and other microbes in skin disease
Sammanfattning : The skin is colonized with a wide range of microbes. Some offer vital protection from colonization and infection with pathogenic strains while others have the capacity to cause or exacerbate disease. LÄS MER
4. Structural and functional studies of Malassezia sympodialis-derived allergens
Sammanfattning : Atopic eczema (AE) is a chronic inflammatory skin disease characterised by pruritic (itchy) skin lesions. The pathogenic mechanisms underlying AE are still unclear, although several factors such as genetic predisposition, a dysfunctional skin barrier, exposure to environmental allergens and skin colonisation with microorganisms appear to be of importance. LÄS MER
5. The role of Malassezia in the pathogenesis of atopic eczema/dermatitis syndrome
Sammanfattning : Atopic eczema/dermatitis syndrome (AEDS) is a chronically relapsing skin disease, which is often associated with elevated levels of serum lgE. The immunopathology of AEDS is at present unclear, but it is known that lymphocytes infiltrating the AEDS lesions are predominantly of the CD4+ T helper phenotype. LÄS MER