Sökning: "myocardial hypertrophy"
Visar resultat 16 - 20 av 37 avhandlingar innehållade orden myocardial hypertrophy.
16. Structural and neurohormonal factors in left ventricular hypertrophy and inhibition of the renin-angiotensin-aldosterone system
Sammanfattning : Hypertensive left ventricular hypertrophy (LVH) is a major risk factor for cardiovascular morbidity and mortality. The aim of this thesis was to study how different neurohormonal systems relate to left ventricular mass, and to study structural and functional cardiovascular changes in hypertensive LVH. LÄS MER
17. Coarctation of the aorta : register and imaging studies
Sammanfattning : Background Coarctation of the aorta (CoA) constitutes 5-8 % of all congenital heart disease (CHD) and is associated with long-term complications such as hypertension (HTN) and left ventricular hypertrophy (LVH). Factors associated with HTN, LVH, and diffuse myocardial fibrosis, are not yet fully explored in this population. LÄS MER
18. Blood Pressure Elevation - Impact on Cardiovascular Structure and Endogenous Fibrinolysis
Sammanfattning : Blood pressure elevation is a major risk factor for cardiovascular events and the risk increases in a dose-dependant manner. It is of importance to identify subjects prone to develop hypertension and adverse cardiovascular remodeling in order to start treatment timely. LÄS MER
19. Impaired glucose tolerance in ischemic heart disease
Sammanfattning : Background: Diabetes mellitus (DM) and impaired glucose tolerance test (IGT) are well known risk factors for cardiovascular disease (CVD). Individuals with IGT, a prediabetic state, are asymptomatic during many years and they often remain undiagnosed until they have developed overt diabetes or cardiovascular complications. LÄS MER
20. Early Cardiovascular Changes of Familial Hypertrophic Cardiomyopathy in the Young
Sammanfattning : Familial hypertrophic cardiomyopathy (HCM) is the most common inherited heart disease, transmitted in an autosomal dominant fashion, i.e. 50% risk for transmission of the disease-causing mutation to each child of the affected family. LÄS MER