Sökning: "human endogenous retroviruses"
Visar resultat 1 - 5 av 14 avhandlingar innehållade orden human endogenous retroviruses.
1. Studies on Human Endogenous Retroviruses (HERVs) with Special Focus on ERV3
Sammanfattning : Human endogenous retroviruses (HERVs) represent approximately 7% of the human genome. This investigation was focused on one particular HERV, ERV3, with the main purpose of characterising its gene expression patterns and genomic distribution of ERV3-like sequences. LÄS MER
2. Human Endogenous Retroviruses: Expression and Evolutionary Relationships
Sammanfattning : The human genome contains genetic elements which are more or less similar to infectious retroviruses. These are called human endogenous retroviruses (HERVs) and are thought to be remnants of infections in the primate lineage. LÄS MER
3. Human Endogenous Retroviruses: Studies on Transcriptional Activity and Genetic Variability
Sammanfattning : Endogenous retroviruses (ERVs) are believed to be remains of germ-line infections that have become fixed in the population. They are involved in pathophysiological conditions, in mechanisms leading to infection protection, and directly in the physiology of the organism by participating in the regulation of cellular gene expression. LÄS MER
4. Studies of human endogenous retroviruses and of the TGF-beta signaling pathway
Sammanfattning : The human genome, in common with all mammalian genomes, carries a high load of inheritable retroviral sequences. We have identified and characterized a novel family of these endogenous retroviruses and have named this family HERV-F as based on the primer binding sites of three members. LÄS MER
5. Transcription, splicing and genetic structure within the human endogenous retroviral HERV-H family
Sammanfattning : Human endogenous retroviruses (HERVs) are remains of ancient retroviral infections of the germ line and constitute as much as 3 % of the human genome. HERVs are genetic elements that potentially may provide important biological functions by several different mechanisms. LÄS MER