Sökning: "angiotensin-converting enzyme inhibitor"
Visar resultat 1 - 5 av 13 avhandlingar innehållade orden angiotensin-converting enzyme inhibitor.
1. Pharmacological interactions between angiotensin-converting enzyme (ACE) inhibitors, bradykinin and nitric oxide
Sammanfattning : Cardiovascular diseases are a major cause of death in Western countries. Angiotensin-converting enzyme (ACE) is as a key enzyme in the renin-angiotensin system involved in the regulation of blood pressure, and water and electrolyte balance in the body. LÄS MER
2. Angiotensin converting enzyme inhibition and aspirin in congestive heart failure
Sammanfattning : Angiotensin-converting enzyme (ACE) inhibitors act by decreasing production of angiotensin II and by potentiating the effects of bradykinin by inhibition of its breakdown. Bradykinin exerts part of its effects via vasodilating prostaglandins. Since the cyclooxygenase (COX)-inhibitors, e.g. LÄS MER
3. Treatment in Dilated Cardiomyopathy : with special emphasis on beta-adrenergic receptor blockade and angiotensin-converting enzyme inhibition
Sammanfattning : Dilated cardiomyopathy (DCM) is a heart muscle disease of unknown origin, characterised by ventricular dilatation and impairment of systolic function. The basic treattnent is medical, according to different pharmacological principles. Evaluation of the severity of the disease and the effects of medication are important for optimal management. LÄS MER
4. Genetic aspects of stroke : association and linkage studies in a northern Swedish population
Sammanfattning : Stroke is a common, multifactorial cardiovascular disease. A stroke event is the result of traditional risk factors (i.e. hypertension, diabetes, smoking), environmental exposures and genetic factors in a complex interplay. LÄS MER
5. Studies on vasoactivity of fibrin(ogen) derived peptides
Sammanfattning : Fibrin accumulation is a common fmding in many pathological conditions, such as in inflammatory processes, e.g. in the lungs in pneumonia or pulmonary insufficiency after sepsis. During lysis of the fibrin by plasmin or leukocyte elastase, peptides are released that may contribute to the pathophysiological changes. LÄS MER