Sökning: "abdominal sepsis"
Visar resultat 11 - 15 av 26 avhandlingar innehållade orden abdominal sepsis.
11. Neutrophils and extracellular traps in abdominal sepsis
Sammanfattning : Abdominal sepsis is associated with changes in systemic inflammation response and coagulation homeostasis. Excessive neutrophil activation is a key regulator in both pro-inflammatory immune response and septic lung injury. Neutrophil-derived DNA may form extracellular traps in response to invading pathogens. LÄS MER
12. On Generation and Function of Neutrophil Extracellular Traps in Abdominal Sepsis
Sammanfattning : AbstractSepsis is a clinical condition that develops when the immune system over-response to an infection. Sepsis can lead to organ failure as well as subsequent death. Sepsis-induced host response involves neutrophil over activation which can lead to self tissue damage. LÄS MER
13. On Neutrophil and Platelet Adhesive Interactions in Septic Lung Injury
Sammanfattning : Sepsis induced by intestinal perforation is a life-threatening illness and is a frequent cause of leukocyte-mediated lung injury. Although antimicrobial therapy has been the central clinical strategy for these patients, the survival rates in such patients are still very low because of their impaired host defense mechanisms. LÄS MER
14. Pancreatitis-Associated Pulmonary Injury
Sammanfattning : In acute pancreatitis (AP), lung injury is an early occuring and important part of the multiple organ dysfunction syndrome. The mechanisms underlying the development of lung injury though still largely remains unclear. LÄS MER
15. Acute pancreatitis - Cellular response and dynamic inflammatory changes
Sammanfattning : Acute pancreatitis (AP) is a common disorder where underlying mechanisms for the local initiating events in the pancreas, the systemic dissemination of the inflammatory response and the remote organ dysfunction still are unclear. The aim of the present study was to further characterize the dynamics and sequence of the immune response in AP, and to determine the cellular origin of the initiating events. LÄS MER