Sökning: "T-Lymphocytes"
Visar resultat 26 - 30 av 231 avhandlingar innehållade ordet T-Lymphocytes.
26. Regulation of activation of NF-κB by Calmodulin in T-lymphocytes
Sammanfattning : Nuclear factor kappa B (NF-kB) is a widely expressed family of transcription factors that are involved in a diverse number of processes. These include inflammation or differentiation, survival or apoptosis, and proliferation or cell cycle arrest. LÄS MER
27. Gene expression analysis of Tec family kinases in B- and T-lymphocytes
Sammanfattning : The microarray technology is a powerful tool used in many different research areas. The technique has been around for more than a decade and has revolutionized the molecular biology field. In this thesis the Affymetrix GeneChip® arrays were used to study the transcriptomes of Tec family kinase mutant mouse models. LÄS MER
28. Role of GABA and GABAA Channels in T lymphocytes and Stem cells
Sammanfattning : GABA (gamma-aminobutyric acid) is best known for its physiological function in the central nervous system. In the brain GABA is the main inhibitory neurotransmitter where it decreases excitability of neurons and neuronal networks. LÄS MER
29. Aberrancies associated with dendritic cells and T lymphocytes in type 1 diabetes
Sammanfattning : The incidence of type 1 diabetes has rapidly increased in the Western world and the age of onset has shifted to younger ages. Type 1 diabetes is a chronic disease caused by destruction of the insulin producing ß-cells in the pancreas leading to severe insulin deficiency. This results in hyperglycaemia and diagnosis of overt type 1 diabetes. LÄS MER
30. T lymphocytes and cytokines in the pathogenesis of experimental autoimmune myasthenia gravis
Sammanfattning : Experimental autoimmune myasthenia gravis (EAMG), induced in different animal species and strains by immunization with nicotinic acetylcholine receptor (AChR) and complete Freund's adjuvant (CFA), represents an experimental model of myasthenia gravis (MG) in humans. Both MG and EAMG are autoimmune diseases mediated by antibodies against AChR of neuromuscular junctions. LÄS MER