Sökning: "Sun Xiao-Feng"
Visar resultat 1 - 5 av 13 avhandlingar innehållade orden Sun Xiao-Feng.
1. Expression of ras and p53, DNA ploidy and 5-phase fraction in human colorectal adenocarcinoma
Sammanfattning : The accumulation of oncogene and anti-oncogene alterations play an important role in the development of colorectal adenocarcinomas. These specific gene alterations cause changes of DNA ploidy and cell proliferation and, in turn, DNA instability might lead to more genetic changes. LÄS MER
2. The CpG island methylator phenotype in colorectal cancer : studies on risk and prognosis
Sammanfattning : Background Colorectal cancer (CRC) is the second most common malignancy in developed countries. The mortality is high, with nearly half of patients dying from the disease. The primary treatment of CRC is surgery, and decisions about additional treatment with chemotherapy are based mainly on tumor stage. LÄS MER
3. Molecular studies of radiotherapy and chemotherapy in colorectal cancer
Sammanfattning : .... LÄS MER
4. Keratinocytes in tissue engineering of human skin: invitro and in vivo studies
Sammanfattning : Full thickness wounds, such as deep burns, need restoration of both the dermal and epidermal layers of the skin. In normal wound healing, re-epithelialization occurs by migration and proliferation of keratinocytes from the wound edges and by differentiation of stem cells from remaining hair follicles. LÄS MER
5. Molecular and Biological Characteristics of Stroma and Tumor Cells in Colorectal Cancer
Sammanfattning : Carcinogenesis is a progressive process involving multiple genetic alterations in tumor cells and complex interactions in the tumor-host microenvironment. To better understand the contribution of molecular alterations in tumor cells and stromal variables to the development of colorectal cancer (CRC) and identify prognostic factors, in this study we examined the clinicopathological and biological significance of stromal variables, including particularly interesting new cysteine-histidine rich protein (PINCH), inflammatory infiltration, angiogenesis and lymphangiogenesis, as well as hRAD50/hMRE11/hNBS1 proteins and hRAD50 mutation in tumor cell in CRC. LÄS MER