Sökning: "Subcutaneous Adipose Tissue"
Visar resultat 1 - 5 av 93 avhandlingar innehållade orden Subcutaneous Adipose Tissue.
1. Adipose tissue as an active organ : blood flow regulation and tissue-specific glucocorticoid metabolism
Sammanfattning : Background: Despite advances in the treatment of atherosclerosis, cardiovascular disease is the leading cause of death worldwide. With the population getting older and more obese, the burden of cardiovascular disease may further increase. LÄS MER
2. Obesity-associated inflammation in adipose tissue
Sammanfattning : Background: Excess body fat, particularly in the visceral depot, is linked to increased mortality and morbidity, including the development of diseases such as type 2 diabetes, cardiovascular disease, and cancer. Chronic low-grade inflammation in adipose tissue may be a key mediator of obesity-associated diseases. LÄS MER
3. Adipose tissue gene expression & candidate genes for obesity
Sammanfattning : Obesity results from an interaction between genetic and environmental factors and is a growing health problem associated with development of insulin resistance, type 2 diabetes and cardiovascular disease. This thesis has focused on investigating the genetic contribution in obesity and insulin resistance by both candidate and global gene approaches with focus on the adipose tissue. LÄS MER
4. Material Modelling of Adipose Tissue for Traffic Injury Prevention
Sammanfattning : Traffic injury is one of the main reasons for traumatic injuries. Obese occupants are among the vulnerable populations with a higher risk of death and severe injuries. Notably, obesity is associated with a thick layer of subcutaneous adipose (fat) tissue. LÄS MER
5. Prolactin receptor expression and prolactin-mediated effects in adipose tissue
Sammanfattning : The aim of this study was to investigate the presence of functional prolactin receptors (PRLRs) in white adipose tissue of mice and humans, and the possible functions mediated via PRLRs. In mouse white adipose tissue, three PRLR mRNA isoforms, L-, S2-, and S3-PRLR, and L-PRLR protein expression were detected. LÄS MER