Sökning: "Neuroinflammation"
Visar resultat 1 - 5 av 121 avhandlingar innehållade ordet Neuroinflammation.
1. Neuroinflammation and pain
Sammanfattning : ABSTRACT Background: Persistent pain that remains long after the physiological trigger has been resolved is a disabling condition. A possible mechanism for the transition from acute physiological pain to persistent pain involves low-grade inflammation in the central nervous system, in which inflammatory-activated astrocytes play a significant role. LÄS MER
2. Neuroinflammation in Alzheimer's disease : Focus on NF-κB and C/EBP transcription factors
Sammanfattning : Alzheimer's disease (AD) is the most common form of dementia among elderly. The disease is characterized by amyloid-β (Aβ) plaques, neurofibrillary tangles, loss of synapses and neurons and chronic neuroinflammation. LÄS MER
3. Neuroinflammation in Alzheimer’s disease and obesity
Sammanfattning : Alzheimer’s disease (AD) and obesity are both major problems in the western world. Although they may appear to have little in common at first glance, they are both characterized by chronic inflammation. Exactly how inflammation affects these disorders is far from clear. LÄS MER
4. Physical exercise as a preventive strategy for disorders affecting the brain. The effect on neuroinflammation
Sammanfattning : Physical activity is associated with lower risks of developing dementia, Parkinson’s disease (PD) and depression. Furthermore, being physically active prior to brain ischemia appears to help the recovery process. LÄS MER
5. Neuroinflammation and amyloid-β in early Alzheimer’s disease. Insight into the earliest events using mouse models
Sammanfattning : Alzheimer’s disease (AD) is the leading cause of dementia and most common neurodegenerative disease worldwide, but there currently exists no effective treatment that can stop nor slow the progression of the disease. The current dogma in the field postulates that the appearance of extracellular amyloid-beta (Aβ) plaques, a histopathological hallmark of the disease, is the trigger for downstream, detrimental events, including neuronal loss, extensive neuroinflammation and cognitive decline. LÄS MER