Avancerad sökning
Visar resultat 1 - 5 av 17 avhandlingar som matchar ovanstående sökkriterier.
1. Liver metastases from colorectal cancer. Different strategies and outcomes
Sammanfattning : Patients with colorectal liver metastases (CRLM) increasingly undergo liver resections. The belief is that the resection or ablation of a tumor, when possible, is the only possibility of a cure. LÄS MER
2. Liver regeneration and function following portal vein occlusion and hepatectomy
Sammanfattning : Background: Portal vein occlusion (PVO) and associating liver partition and portal vein ligation for staged hepatectomy (ALPPS) are two strategies employed to render patients with unresectable liver tumours resectable by increasing the size of the future liver remnant (FLR). There is still limited knowledge about several aspects of PVO and most aspects of ALPPS. LÄS MER
3. Outcome after liver resection : epidemiological and clinical studies with special focus on the role of post-hepatectomy liver failure
Sammanfattning : Background: Liver surgery has undergone significant developments over the last three decades. However, population-based data are scarce, and there is limited knowledge of the impact on mortality and possible treatment modalities of a feared complication following hepatectomy, post-hepatectomy liver failure (PHLF). LÄS MER
4. Colorectal Liver Metastases – Different Aspects on Treatment with Associated Liver Partition and Portal Vein Ligation for Staged Hepatectomy and on Portal Vein Occlusion
Sammanfattning : Introduction: For patients with colorectal liver metastases (CRLM), the only treatment with a possibility for long-term survival and cure is radical resection. The majority of patients are at the time of diagnosis not assessed as resectable because they have advanced disease in the liver or unresectable extrahepatic disease or are too frail to withstand liver surgery. LÄS MER
5. Prevention of liver cancer in chronic liver disease : an experimental study of sodium selenite and rat hepatocarcinogenesis
Sammanfattning : Selenium treatment in supranutritional but subtoxic doses has previously been shown to inhibit cell proliferation in preneoplastic lesions in a rat liver carcinogenesis model. The mechanisms are not known, but thioredoxin reductase (TrxR1), a seleno-enzyme essential for maintaining intracellular redox status and cellular defence against oxidative stress might be involved. LÄS MER