Sökning: "Exercise tolerance"

Visar resultat 1 - 5 av 41 avhandlingar innehållade orden Exercise tolerance.

1. 1. Exercising on the edge: mitochondrial and metabolic responses to intense training

   Författare :Mikael Flockhart; Filip J Larsen; Eva Blomstrand; Nikolai Baastrup Nordsborg; Gymnastik- och idrottshögskolan; []
   Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; Exercise; training; mitochondria; performance; glucose tolerance; proteins; oxygen uptake; metabolism; cycling; Medicin Teknik; Medicine Technology;

   Sammanfattning : Exercise and exercise training induces several physiological adaptations that increase the oxidative capacity of the muscles and improve glucose regulation. While the positive metabolic adaptations and effects on glucose regulation after exercise and exercise training have been extensively studied, negative outcomes have not. LÄS MER

3. 2. Exercise in patients with chronic heart failure. With emphasis on peripheral muscle training, hydrotherapy and type 2 diabetes mellitus

   Författare :Åsa Cider; Göteborgs universitet; []
   Nyckelord :Exercise tolerance; Ejection fraction; Metabolic function; Muscle function; Oxygen uptake; Physical therapy techniques; Quality of life; Rehabilitation; Stroke volume; Water immersion;

   Sammanfattning : Chronic heart failure (CHF) is not uncommon in developed countries with a growing ageing population. CHF is a chronic syndrome, which markedly impact negatively on the patient s life situation. Considerable impaired physical function and reduced health related quality of life is often present. LÄS MER

4. 3. Environmental Factors of Importance in Myasthenia Gravis : Emphasis on Physical Activity

   Författare :Elisabet Westerberg; Anna Rostedt Punga; Anne-Marie Landtblom; Chantal Tallaksen; Uppsala universitet; []
   Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; Myasthenia Gravis; MG subgroups; Environmental factors; Lifestyle; Physical activity; Exercise; Clinical Neurophysiology; Klinisk neurofysiologi;

   Sammanfattning : Myasthenia Gravis (MG) is an autoimmune disease caused by antibodies directed against proteins at the neuromuscular junction (NMJ). The disturbed neuromuscular transmission induces fatigable weakness in skeletal muscles. LÄS MER

5. 4. Inflammation and lifestyle in cardiovascular medicine

   Författare :Jonas Andersson; Kurt Boman; Jan-Håkan Jansson; Lars Johansson; Joep Perk; Umeå universitet; []
   Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; C-reactive protein; cardiovascular disease; stroke; atrial fibrillation; lifestyle; interleukin-6; tumor necrosis factor-α; exercise; physical activity; obesity; the metabolic syndrome; fibrinolysis; Cardiology; Kardiologi; kardiologi; Cardiology;

   Sammanfattning : Despite major advances in the treatment and prevention of atherosclerosis the last several decades, cardiovascular disease still accounts for the majority of deaths in Sweden. With the population getting older, more obese and with rising numbers of diabetics, the cardiovascular disease burden may increase further in the future. LÄS MER

7. 5. Lifestyle and glycaemic control before and after the onset of type 2 diabetes

   Författare :Robert Koivula; Genetisk och molekylär epidemiologi; []
   Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; Type 2 Diabetes; Prediabetes; Obesity; Beta-cell Function; Insulin Sensitivity; Physical Activity; Exercise; Diet; Lifestyle; Ectopic Fat; Genetic Epidemiology; Genetic Risk; Biomarker; Precision Medicine;

   Sammanfattning : Type 2 diabetes (T2D) is a complex disease with widespread physiological insults to the regulation of metabolic homeostasis, above all glycaemic regulation. The pathogenesis of T2D and its progression is broadly understood to be through a gradual decrease in peripheral insulin sensitivity, a compensatory rise in insulin secretion, and a gradual decline in beta-cell function, resulting in glycaemic dysregulation and eventual T2D. LÄS MER