Sökning: "A Schölin"

Hittade 3 avhandlingar innehållade orden A Schölin.

  1. 1. Getting a Say : Bringing patients’ views on benefit-risk into medical approvals

    Författare :Karin Schölin Bywall; Ulrik Kihlbom; Mats G. Hansson; Jorien Veldwijk; Peter Mol; Uppsala universitet; []
    Nyckelord :Educational material; Medical product lifecycle; Patient preferences; Regulatory marketing authorisations; Rheumatoid arthritis;

    Sammanfattning : The focus of this thesis is a new quantitative approach to consider patient preferences on benefits and risks in medical approvals. The overall aim of this thesis was to explore how patient preference information may be relevant to regulatory marketing authorisation decisions. LÄS MER

  3. 2. Etnisk mångfald som organisationsidé. Chefs- och personalpraktiker i äldreomsorgen

    Författare :Tobias Schölin; Malmö högskola; []
    Nyckelord :Diversity; Ethnic diversity; Diversity management; Leadership; Management and staff practices; Ethnicity; Social construction; Eldercare; Everyday practices; Organisation theory;

    Sammanfattning : The dissertation’s research context is organisation, ethnic diversity and healthcare. The focus is on healthcare managers’ relations to the organisational idea of diversity and the problematisation of how ethnicity is constructed by eldercare staff. LÄS MER

  4. 3. Diabetes in Young Adults : Remission, β-cell function and markers of inflammation

    Författare :Anna Schölin; Elisabeth Björk; Anders Karlsson; Jan Åman; Uppsala universitet; []
    Nyckelord :MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; Internal medicine; type 1 diabetes; young adults; remission; β-cell function; islet antibodies; gender; CRP; Invärtesmedicin; Internal medicine; Invärtesmedicin; Internal Medicine; invärtesmedicin;

    Sammanfattning : Type 1 diabetes is caused by immuno-mediated β-cell destruction leading to insulin deficiency and hyperglycaemia. The decline in β-cell function and the clinical course after diagnosis vary. Whether the process of destruction of the β-cells is associated with markers of a non-specific inflammatory response is unknown. LÄS MER