Sökning: "åderförkalkning"
Visar resultat 1 - 5 av 50 avhandlingar innehållade ordet åderförkalkning.
1. Assessment of predicting blood flow and atherosclerosis in the aorta and renal arteries
Sammanfattning : Cardiovascular diseases (CVD) are the most common cause of death in large parts of the world. Atherosclerosis (AS) has a major part in most CVDs. AS is a slowly developingdisease which is dependent on multiple factors such as genetics and life style (food, smoking, and physical activities). LÄS MER
2. Ultrasound and atherosclerosis. Evaluation of methods, risk factors and intervention
Sammanfattning : The importance of studying cardiovascular disease before the process leads to hard end-points has been recognised, and methods to quantify early atherosclerosis have been taken in use in many research centres around the world. An ultrasound method with a computer assisted image analysing system was developed in co-operation with the Wallenberg Laboratory for Cardiovascular Research and Chalmers University of Technology in Gothenburg, Sweden. LÄS MER
3. Regulatory Immune Responses and Repair Mechanisms in Atherosclerosis
Sammanfattning : Atherosclerosis is a chronic inflammatory disease characterized by the formation of lipid rich plaques in the arterial wall. Rupture of a plaque results in clinical manifestations such myocardial infarction or stroke. LÄS MER
4. Angiotensin-Converting Enzyme : Effects of Smoking and Other Risk Factors for Cardiovascular Diseases
Sammanfattning : Cardiovascular diseases (CVDs) are the most common cause of death in Western countries. Smoking, hypertension, diabetes mellitus and hypercholesterolemia are considered as major risk factors. However, the underlying mechanisms by which these factors cause CVDs are not entirely clear. LÄS MER
5. Endothelium-derived Vasoactive Factors and Leukocyte-derived Inflammatory Mediators. Studies in subjects with or at risk for vascular disease
Sammanfattning : This study was undertaken to evaluate relationships between endothelial function (assessed as mediators of endothelium-derived vasoactive factors such as nitric oxide [NO], prostacyclin [PGI2] and endothelin [ET-1] ), and risk factors for atherosclerosis, as well as to study possible relationships between endothelial function and leukocyte activation (indicated by measurements of leukocyte-derived inflammatory mediators) in subjects with or at risk for atherosclerosis. In subjects with asymptomatic atherosclerosis we found inverse relationships between cyclic guanosine monophosphate (cGMP) in platelets (mediator of NO activity) and both serum total cholesterol and plasma ET-1 suggesting that NO-mediated vasodilation may be impaired in hypercholesterolemia and that maintenance of vascular tone is achieved by a balance between NO and ET-1. LÄS MER
